Misdiagnosis of diabetic oculomotor palsy _3806

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Misdiagnosis of diabetic oculomotor palsy
 
 
Positively charged amino sodium channels with the inside of the gate side of the membrane phospholipid molecules with the negatively charged phosphate -binding ,You are not allowed to view links. Register or Login, resulting in changes in the structure of sodium channels , sodium channel inactivation gate closed, can not produce action potentials ,You are not allowed to view links. Register or Login, nerve impulse conduction termination , Which leads to partial paralysis . Glucocorticoids in the maintenance of normal blood glucose levels and liver and muscle glycogen content plays an important role . Although the role of anti-inflammatory such as prednisone , but also the liver , muscle glycogenolysis ,You are not allowed to view links. Register or Login, elevated blood glucose . Thus , after injection of lidocaine , its local levator nerve palsy and prednisone ascending blood glucose,You are not allowed to view links. Register or Login, and diabetes increased the cranial nerve lesions , causing ptosis, ophthalmoplegia . 2.3 The admissions prompted doctors should diagnose detailed history , complete the relevant checks,You are not allowed to view links. Register or Login, open your mind, a wealth of medical knowledge related to minimize such problems. Tip diabetes can not use the partial closure of glucocorticoid treatment, to avoid worse.